arXiv:2603. 14169v2 Announce Type: replace-cross Abstract: Average treatment effects (ATE) and conditional average treatment effects (CATE) are foundational causal estimands, but they target changes in expected outcomes and can miss treatment-induced changes in the shape of outcome distributions.
By Amir Saki, Usef Faghihi
arXiv:2606. 00754v1 Announce Type: cross Abstract: We introduce causal density functions: Radon-Nikodym derivatives that compare interventional laws to observational laws and therefore act as local density ratios for causal effects.
By Sridhar Mahadevan
arXiv:2604. 23904v3 Announce Type: replace-cross Abstract: Synthetic tabular data are often evaluated by distributional similarity, privacy distance, or train-on-synthetic-test-on-real predictive performance, but these criteria do not ensure validity for causal inference.
By Yichen Xu
arXiv:2606. 19610v1 Announce Type: cross Abstract: Recent work on Kan-Do-Calculus (KDC) has established that the boundary between passive observation and active intervention in causal inference is a category-theoretic bi-adjunction, with interventions modeled by left Kan extensions and conditioning by right Kan extensions.
By Sridhar Mahadevan
arXiv:2604. 16763v3 Announce Type: replace Abstract: Causal inference from electronic health records (EHR) is fundamentally limited by unmeasured confounding: critical clinical states such as frailty, goals of care, and mental status are documented in free-text notes but absent from structured data.
By Lei Liu, Jialin Chen, Kathy Macropol
arXiv:2605. 03268v2 Announce Type: replace-cross Abstract: Here we introduce Partially Observed Structural Causal Models (POSCMs) as an extension of structural causal models (SCMs) to settings where upstream contexts co-determine both the interaction structure and downstream mechanisms on observed variables.
By Turan Orujlu, Jordan Matelsky, Martin V. Butz, Charley M. Wu, Konrad P. Kording
arXiv:2505. 17961v4 Announce Type: replace-cross Abstract: Causal inference typically assumes centralized access to individual-level data.
By R\'emi Khellaf, Aur\'elien Bellet, Julie Josse
arXiv:2608. 00657v1 Announce Type: cross Abstract: Causal inference usually concerns a scalar treatment, yet in many problems the treatment is unstructured: a text, an image, or a sequence of clinical decisions.
By Kevin Christian Wibisono, Yixin Wang
arXiv:2607. 26521v1 Announce Type: new Abstract: Conventional subgroup analyses can yield unstable and difficult-to-interpret conclusions, especially in observational biomedical data where each individual is observed under only one exposure state, true individual treatment effects are unavailable, and causal structure is uncertain.
By Vasundhara Acharya, Bulent Yener
arXiv:2608. 02877v1 Announce Type: new Abstract: Causal discovery recovers directed structure from observational data and is increasingly used in clinical settings to support mechanism reasoning and fairness audits of predictive models.
By Nitish Nagesh, Elahe Khatibi, Thomas Dean Hughes, Mahdi Bagheri, Pratik Gajane, Amir M. Rahmani
arXiv:2608. 12555v1 Announce Type: new Abstract: Predictive explanation methods attribute a model output; they do not, by themselves, attribute an intervention effect on the real-world outcome.
By Michael Georgiades, Charalambia Varnava
arXiv:2608. 05244v1 Announce Type: cross Abstract: We developed a unified covariate-adjusted causal inference framework for estimating the desirability of outcome ranking (DOOR) probability for benefit-risk evaluation in randomized trials and observational studies.
By Yuan Feng, Shiyu Shu, Yixin Fang, Ionut Bebu, Toshimitsu Hamasaki, Scott Evans, Guoqing Diao