arXiv:2608. 01352v1 Announce Type: new Abstract: Estimating causal effects from real-world spatiotemporal data is challenging due to hidden confounders and interference.
arXiv:2510. 08762v2 Announce Type: replace Abstract: Causal inference in spatial domains faces two intertwined challenges: (1) unmeasured spatial factors, such as weather, air pollution, or mobility, that confound treatment and outcome, and (2) interference from nearby treatments that violate standard no-interference assumptions.
arXiv:2609.40051v1 Announce Type: new Abstract: Estimating causal effects from observational data is central to science and policy, but the effects are not identified when confounders are unmeasured....
arXiv:2604. 23904v3 Announce Type: replace-cross Abstract: Synthetic tabular data are often evaluated by distributional similarity, privacy distance, or train-on-synthetic-test-on-real predictive performance, but these criteria do not ensure validity for causal inference.
arXiv:2608. 08064v1 Announce Type: new Abstract: Learning fine-grained spatial patterns from coarse-resolution data is challenging, especially in causal settings where high-resolution effects must be inferred from aggregated interventions and outcomes.
arXiv:2606. 21806v2 Announce Type: replace Abstract: Deep generative models reproduce the observational distribution of their training data, inheriting any spurious associations it contains.
arXiv:2608. 08288v1 Announce Type: new Abstract: Estimating counterfactual outcomes over time from longitudinal observational data is central to clinical decision support.
The paper studies when joint-embedding predictive architectures (JEPAs) can recover underlying causal states from high‑dimensional observations. It introduces a latent variable model where observations arise from causal states with action‑conditioned dynamics, and proposes an information‑theoretic objective that maximizes conditional likelihood while preserving state entropy. The authors prove identifiability conditions—particularly sufficient action‑induced variation—and instantiate the objective as an action‑modulated Gaussian additive‑noise model (A‑JEPA), demonstrating theoretical and empirical success in synthetic and visual benchmarks.
The paper introduces a control‑variable framework for deep neural networks to mitigate omitted variable bias, particularly shortcut learning where covariates like demographics influence predictions. It refits the final layer of a pre‑trained network using cross‑fitting with ridge penalisation, orthogonalises covariate effects, and marginalises predictions over covariate distributions to achieve unbiased, interpretable results. Experiments on simulated images and neuroimaging data show consistent estimation of true effects and performance close to models trained on unconfounded data.
The paper tackles the problem of estimating causal effects when an unobserved confounder is present. It assumes a single, possibly multi‑dimensional proxy variable for the confounder and knowledge of the mechanism that generates this proxy. Under the Single Proxy Identifiability of Causal Effects (SPICE) assumption, the authors prove that the error mechanism is complete and causal effects are identifiable, extending prior proxy‑based results to continuous, multi‑dimensional settings and more flexible functional forms. They also introduce SPICE‑Net, a neural‑network‑based framework for estimating causal effects applicable to both discrete and continuous treatments.
arXiv:2606. 17010v1 Announce Type: new Abstract: Heterogeneous Treatment Effect (HTE) identification is crucial to explain the impact of an intervention and optimize our policies accordingly.
arXiv:2609.23753v1 Announce Type: cross Abstract: Generative world models aim to predict future states conditioned on actions, where action controllability is fundamental for reliable dynamics modeli...
arXiv:2510. 21457v2 Announce Type: replace Abstract: Estimating heterogeneous treatment effects in network settings is complicated by interference, meaning that the outcome of an instance can be influenced by the treatment status of others.