arXiv:2603. 14169v2 Announce Type: replace-cross Abstract: Average treatment effects (ATE) and conditional average treatment effects (CATE) are foundational causal estimands, but they target changes in expected outcomes and can miss treatment-induced changes in the shape of outcome distributions.
By Amir Saki, Usef Faghihi
arXiv:2606. 00754v1 Announce Type: cross Abstract: We introduce causal density functions: Radon-Nikodym derivatives that compare interventional laws to observational laws and therefore act as local density ratios for causal effects.
By Sridhar Mahadevan
arXiv:2604. 23904v3 Announce Type: replace-cross Abstract: Synthetic tabular data are often evaluated by distributional similarity, privacy distance, or train-on-synthetic-test-on-real predictive performance, but these criteria do not ensure validity for causal inference.
By Yichen Xu
arXiv:2606. 19610v1 Announce Type: cross Abstract: Recent work on Kan-Do-Calculus (KDC) has established that the boundary between passive observation and active intervention in causal inference is a category-theoretic bi-adjunction, with interventions modeled by left Kan extensions and conditioning by right Kan extensions.
By Sridhar Mahadevan
arXiv:2604. 16763v3 Announce Type: replace Abstract: Causal inference from electronic health records (EHR) is fundamentally limited by unmeasured confounding: critical clinical states such as frailty, goals of care, and mental status are documented in free-text notes but absent from structured data.
By Lei Liu, Jialin Chen, Kathy Macropol
arXiv:2605. 03268v2 Announce Type: replace-cross Abstract: Here we introduce Partially Observed Structural Causal Models (POSCMs) as an extension of structural causal models (SCMs) to settings where upstream contexts co-determine both the interaction structure and downstream mechanisms on observed variables.
By Turan Orujlu, Jordan Matelsky, Martin V. Butz, Charley M. Wu, Konrad P. Kording